Hochrainer, Karin

Publications

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selected publications

Post-ischemic ubiquitination at the postsynaptic density reversibly influences the activity of ischemia-relevant kinases. Communications biology. 2024 Academic Article GET IT
Post-ischemic ubiquitination at the postsynaptic density reversibly influences the activity of ischemia-relevant kinases. 2023 GET IT
Elevated post-ischemic ubiquitination results from suppression of deubiquitinase activity and not proteasome inhibition. Cellular and molecular life sciences : CMLS. 2020 Academic Article GET IT
Times cited: 7
Tau induces PSD95-neuronal NOS uncoupling and neurovascular dysfunction independent of neurodegeneration. Nature neuroscience. 2020 Academic Article GET IT
Times cited: 61
Dietary salt promotes cognitive impairment through tau phosphorylation. Nature. 2019 Academic Article GET IT
Times cited: 140
Cerebral ischemia induces the aggregation of proteins linked to neurodegenerative diseases. Scientific reports. 2018 Academic Article GET IT
Times cited: 59
The ubiquitin ligase HERC3 attenuates NF-κB-dependent transcription independently of its enzymatic activity by delivering the RelA subunit for degradation. Nucleic acids research. 2015 Academic Article GET IT
Times cited: 21
SUMO2/3 is associated with ubiquitinated protein aggregates in the mouse neocortex after middle cerebral artery occlusion. Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. 2014 Academic Article GET IT
Times cited: 25
Progranulin deficiency promotes post-ischemic blood-brain barrier disruption. The Journal of neuroscience : the official journal of the Society for Neuroscience. 2013 Academic Article GET IT
Times cited: 75
Lipoprotein receptor-related protein-6 protects the brain from ischemic injury. Stroke. 2013 Academic Article GET IT
Times cited: 20
Site-specific phosphorylation of the p65 protein subunit mediates selective gene expression by differential NF-κB and RNA polymerase II promoter recruitment. The Journal of biological chemistry. 2012 Academic Article GET IT
Times cited: 57
Regulation of nuclear factor κB (NF-κB) transcriptional activity via p65 acetylation by the chaperonin containing TCP1 (CCT). PloS one. 2012 Academic Article GET IT
Times cited: 29
Reperfusion rather than ischemia drives the formation of ubiquitin aggregates after middle cerebral artery occlusion. Stroke. 2012 Academic Article GET IT
Times cited: 46
Monoubiquitination of nuclear RelA negatively regulates NF-κB activity independent of proteasomal degradation. Cellular and molecular life sciences : CMLS. 2012 Academic Article GET IT
Times cited: 22
Crosstalk between the NF-kappaB activating IKK-complex and the CSN signalosome. Journal of cellular and molecular medicine. 2009 Academic Article GET IT
Times cited: 24
Highly homologous HERC proteins localize to endosomes and exhibit specific interactions with hPLIC and Nm23B. Cellular and molecular life sciences : CMLS. 2008 Academic Article GET IT
Times cited: 22
Nuclear factor-kappaB activation and postischemic inflammation are suppressed in CD36-null mice after middle cerebral artery occlusion. The Journal of neuroscience : the official journal of the Society for Neuroscience. 2008 Academic Article GET IT
Times cited: 96
Phosphatidylethanolamine critically supports internalization of cell-penetrating protein C inhibitor. The Journal of cell biology. 2007 Academic Article GET IT
Times cited: 39
Hypo-phosphorylation leads to nuclear retention of NF-kappaB p65 due to impaired IkappaBalpha gene synthesis. FEBS letters. 2007 Academic Article GET IT
Times cited: 15
Ubiquitylation within signaling pathways in- and outside of inflammation. Thrombosis and haemostasis. 2007 Information Resource GET IT
Times cited: 12
The human HERC family of ubiquitin ligases: novel members, genomic organization, expression profiling, and evolutionary aspects. Genomics. 2005 Academic Article GET IT
Times cited: 51

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Research

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Co-investigator Network

Funding awarded

Tau-induced postsynaptic dysfunction in tauopathy models awarded by Cure Alzheimer’s Fund Principal Investigator 2023 - 2024
Role of CaMKII-alpha ubiquitination in ischemic stroke awarded by National Institute of Neurological Disorders & Stroke Principal Investigator 2018 - 2024
Harriman Foundation Bridge Grant Principal Investigator 2015 - 2016
President’s Council of Cornell Women Grant Principal Investigator 2014 - 2015
American Heart Association Scientist Development Grant 10DSG2600298 Principal Investigator 2010 - 2013

Alzheimer Pathology and Neurovascular Dysfunction awarded by National Institute of Neurological Disorders & Stroke Co-Investigator 2023 - 2027

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Background

education and training

Ph.D., Medical University of Vienna (Austria) 2005
M.S., University of Vienna (Austria) 2000

Contact

full name

Karin Hochrainer

primary email

kah2015@med.cornell.edu

VIVO Weill Cornell Medical College

Karin Hochrainer Assistant Professor of Neuroscience

Publications

selected publications

Research

Funding awarded

Background

education and training

Contact

full name

primary email

Phone

Publications

selected publications

Research

Funding awarded

Background

education and training

Contact

full name

primary email